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Please be aware that this old REACH registration data factsheet is no longer maintained; it remains frozen as of 19th May 2023.
The new ECHA CHEM database has been released by ECHA, and it now contains all REACH registration data. There are more details on the transition of ECHA's published data to ECHA CHEM here.
Diss Factsheets
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EC number: 231-765-0 | CAS number: 7722-84-1
- Life Cycle description
- Uses advised against
- Endpoint summary
- Appearance / physical state / colour
- Melting point / freezing point
- Boiling point
- Density
- Particle size distribution (Granulometry)
- Vapour pressure
- Partition coefficient
- Water solubility
- Solubility in organic solvents / fat solubility
- Surface tension
- Flash point
- Auto flammability
- Flammability
- Explosiveness
- Oxidising properties
- Oxidation reduction potential
- Stability in organic solvents and identity of relevant degradation products
- Storage stability and reactivity towards container material
- Stability: thermal, sunlight, metals
- pH
- Dissociation constant
- Viscosity
- Additional physico-chemical information
- Additional physico-chemical properties of nanomaterials
- Nanomaterial agglomeration / aggregation
- Nanomaterial crystalline phase
- Nanomaterial crystallite and grain size
- Nanomaterial aspect ratio / shape
- Nanomaterial specific surface area
- Nanomaterial Zeta potential
- Nanomaterial surface chemistry
- Nanomaterial dustiness
- Nanomaterial porosity
- Nanomaterial pour density
- Nanomaterial photocatalytic activity
- Nanomaterial radical formation potential
- Nanomaterial catalytic activity
- Endpoint summary
- Stability
- Biodegradation
- Bioaccumulation
- Transport and distribution
- Environmental data
- Additional information on environmental fate and behaviour
- Ecotoxicological Summary
- Aquatic toxicity
- Endpoint summary
- Short-term toxicity to fish
- Long-term toxicity to fish
- Short-term toxicity to aquatic invertebrates
- Long-term toxicity to aquatic invertebrates
- Toxicity to aquatic algae and cyanobacteria
- Toxicity to aquatic plants other than algae
- Toxicity to microorganisms
- Endocrine disrupter testing in aquatic vertebrates – in vivo
- Toxicity to other aquatic organisms
- Sediment toxicity
- Terrestrial toxicity
- Biological effects monitoring
- Biotransformation and kinetics
- Additional ecotoxological information
- Toxicological Summary
- Toxicokinetics, metabolism and distribution
- Acute Toxicity
- Irritation / corrosion
- Sensitisation
- Repeated dose toxicity
- Genetic toxicity
- Carcinogenicity
- Toxicity to reproduction
- Specific investigations
- Exposure related observations in humans
- Toxic effects on livestock and pets
- Additional toxicological data
Toxicity to terrestrial plants
Administrative data
Link to relevant study record(s)
Description of key information
Key value for chemical safety assessment
Additional information
The terrestrial compartment is not the primary environmental compartment affected by environmental emissions of the substance. Hydrogen peroxide occurs naturally in the environment, for example in rain water. In addition, hydrogen peroxide is continuously generated in physiological processes of the plants, for example in the mitochondria (e.g. Rasmusson et al. 1998). A complex system of enzymes, such as superoxide dismutase, ascorbate oxidase, catalase and glutathione peroxidase, exists in plants that scavenges reactive oxygen species like hydrogen peroxide (e.g. Mittler et al. 2004). There is evidence that hydrogen peroxide activates defence responses of plants to various environmental stresses (e.g. Bhattachrjee 2005, Neill et al. 2002). It has also been suggested that hydrogen peroxide at lower doses acts as a signal molecule during plant growth and development. From this it is concluded that terrestrial plants can cope with the levels of exposure to hydrogen peroxide that are to be expected from the identified uses of the substance. No additional testing is deemed necessary.
References
Bhattachrjee S. 2005. Reactive oxygen species and oxidative burst: roles in stress, senescence and signal transduction in plant. Current Science 89(7): 1113-1121.
Mittler R, Vanderauwera S, Gollery M and van Breusegem F. 2004. Reactive oxygen gene network of plant. Trends in Plant Science 9(10): 490-498.
Neill SJ, Desikan R and Hancock JT. 2002. Hydrogen peroxide signalling. Current Opinion in Plant Biology 5(5): 388-395.
Rasmusson AG, Heiser VV, Zabaleta E, Brennicke A, Grohmann L. 1998. Physiological, biochemical and molecular aspects of mitochondrial complex I in plants. Biochim Biophys Acta 1364(2): 101-111.
Information on Registered Substances comes from registration dossiers which have been assigned a registration number. The assignment of a registration number does however not guarantee that the information in the dossier is correct or that the dossier is compliant with Regulation (EC) No 1907/2006 (the REACH Regulation). This information has not been reviewed or verified by the Agency or any other authority. The content is subject to change without prior notice.
Reproduction or further distribution of this information may be subject to copyright protection. Use of the information without obtaining the permission from the owner(s) of the respective information might violate the rights of the owner.